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In addition, in PC3 cell xenograft tumor tissues, C10 treatment inhibited tumor development and upregulated PKCδ. These conclusions advise that C10 treatment method induces the PKCδ/JNK pathway, therefore activating Caspase-3 and inducing the cleavage of PARP and gasdermin E to execute apoptosis and mobile-lytic pyroptosis in prostate cancer cell